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Based on this broad overview it is not dif cult to construct a simplistic conceptual model from which evidence for the links between hierarchic levels of impact of pollution on sh can be examined. Whilst the model provides a useful framework around which the review can be developed, it is important to remember that there are many other factors, both biotic and abiotic, which affect an organism throughout its life. However, to incorporate these parameters into the conceptual model would make it too complicated to identify the potential mechanistic links between the levels of response. This book presents a status report on the published relationships between each of the hierarchic levels of response to pollution from molecular to population and economic. It identi es and con rms links where possible, whether these are real or perceived, using literature on sh and, where necessary, invertebrates and other model organisms. The following chapters describe the impacts of pollution on and between each level of organisation in much more detail, highlighting links where applicable and homeostasis.

This found some objections in the November 1986 meeting, since it was not in line with present legislation in some countries. A footnote was added pointing to this fact. Then the draft recommendation was agreed at the WP1 meeting in January 1987. 43 The drafting group presented a very focussed draft GSM 02.15 Circulation of Mobile Stations . 44 It recommended that:

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results (Fairburn et al, 1999) The heritability of AN was estimated to be 76% in another population-based twin sample (Klump et al, 2001) and 58% in a bivariate analysis of AN and major depression (Wade et al, 2000) The magnitude of the genetic contribution to AN remains unresolved Large-scale twin studies are needed to de ne the extent and nature of the genetic and environmental contributions to the aetiology of AN Three clinically ascertained twin pair studies of BN (Fichter & Noegal, 1990; Hsu et al, 1990; Treasure & Holland, 1989) and two population-based twin studies of BN (Wade et al, 1999; Bulik et al, 1998; Kendler et al, 1991, 1995) have been conducted and have consistently demonstrated signi cant genetic contributions in the liability to BN.

Reanalysis of the data from the twin studies of BN produced estimates of heritabilty ranging from 31% to 83% (Bulik et al, 2000) In general non-shared environmental effects were shown to account for the remaining variance in liability to BN The magnitude of the contribution of shared environmental effects is unclear but in the majority of the twin studies it appears to be less important than additive genetic effects and non-shared environmental effects (Bulik et al, 2000) Several of the symptoms, behaviours and attitudes associated with disordered eating have been shown to be heritable in different populations of twins These continuous traits are assessed using psychometric questionnaires, such as the Eating Disorder Inventory (EDI; Garner et al, 1984) and the Eating Disorders Examination (EDE; Fairburn & Cooper, 1993).

The Drive for Thinness subscale of the EDI, was shown to be heritable in one twin population (Holland et al, 1988), and in another twin study several EDI subscales showed heritabilities ranging from 28% to 52% (Rutherford et al, 1993) Heritabilities of 46% and 70% have been reported for binge eating and self-induced vomiting respectively (Sullivan et al, 1998) There is evidence of age-related differences in genetic and environmental in uences on these traits Marked differences in heritabilities for EDI subscales have been reported for a preadolescent (aged 11 years) and an adolescent (aged 17 years) twin sample from the same population (Klump et al, 2000) The contribution of additive genetic effects for the EDI subscales was greater in the adolescent twin sample than in the preadolescent group.

Drug courts evolved out of earlier unsuccessful, often fragmented, efforts to link drug offenders with treatment services. There are over 1000 drug courts in the planning and implementation phases in the United States and beyond (National Drug Court Institute, 2001). They are dedicated courtrooms that provide judicially-monitored treatment, drug testing and other services to drug-involved offenders (Belenko, 1998, p. 4). These courts seek to craft custom-designed sentences for drug offenders that

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